There are hypochodriacs out there who latch onto this concept of programmed death – especially when scientists come along and verify their assumptions, no matter how off the mark they are:
Ann N Y Acad Sci. 2002 Apr;959:214-37. Related Articles, Links
Programmed death phenomena: from organelle to organism.
Skulachev VP.Department of Bioenergetics, A. N. Belozersky Institute of Physico-Chemical Biology, Moscow State University, Moscow 119899, Russia. skulach@genebee.msu.su
Programmed death phenomena appear to be inherent not only in living cells (apoptosis), but also in subcellular organelles (e.g., self-elimination of mitochondria, called mitoptosis), organs (organoptosis), and even whole organisms (phenoptosis). In all these cases, the “Samurai law of biology”–it is better to die than to be wrong–seems to be operative. The operation of this law helps complicated living systems avoid the risk of ruin when a system of lower hierarchic position makes a significant mistake. Thus, mitoptosis purifies a cell from damaged and hence unwanted mitochondria; apoptosis purifies a tissue from unwanted cells; and phenoptosis purifies a community from unwanted individuals. Defense against reactive oxygen species (ROS) is probably one of the primary evolutionary functions of programmed death mechanisms. So far, it seems that ROS play a key role in the mito-, apo-, organo-, and phenoptoses, which is consistent with Harman’s theory of aging. Here a concept is described that tries to unite Weismann’s hypothesis of aging as an adaptive programmed death mechanism and the generally accepted alternative point of view that considers aging as an inevitable result of accumulation in an organism of occasional injuries. It is suggested that injury accumulation is monitored by a system(s) actuating a phenoptotic death program when the number of injuries reaches some critical level. The system(s) in question are organized in such a way that the lethal case appears to be a result of phenoptosis long before the occasional injuries make impossible the functioning of the organism. It is stressed that for humans these cruel regulations look like an atavism that, if overcome, might dramatically prolong the human life span…
Upon further examination, we discover other facets of programmed cell death insofar as its origin and proper application. Here are two extracts from Wikipedia:
Phenoptosis (pheno – showing or demonstrating, ptosis – programmed death) signifies the phenomenon of programmed death of an organism, ie that an organism’s biology includes features that under certain circumstances will cause it to rapidly degenerate and die off. Phenoptosis is a common feature of living species, whose ramifications for humans is still being explored.
A common route for phenoptosis is breakdown of glucocorticoid regulation and inhibition, leading to massive excess of these corticosteroids in the body. [1]
The process affects many species, from yeast to salmon…
Programmed cell-death (PCD) is death of a cell in any form, mediated by an intracellular program.[1] In contrast to necrosis, which is a form of cell-death that results from acute tissue injury and provokes an inflammatory response, PCD is carried out in a regulated process which generally confers advantage during an organism’s life-cycle. PCD serves fundamental functions during both plant and metazoa (multicellular animals) tissue development. * Apoptosis or Type I cell-death* Autophagic or Type II cell-death ( cytoplasmic: characterized by the formation of large vacuoles which eat away organelles in a specific sequence prior to the nucleus being destroyed.)[2]Besides these two types of PCD, other pathways have been discovered.[3] Called “non-apoptotic programmed cell-death” (or “caspase-independent programmed cell-death” or “necrosis-like programmed cell-death”) these alternative routes to death are as efficient as apoptosis and can function as either backup mechanisms or the main type of PCD.
Other forms of programmed cell death include anolkis, almost identical to apoptosis except in its induction; cornification, a form of cell death exlusive to the eyes; excitotoxicity and Wallerian degeneration.
Plant cells undergo particular processes of PCD which are similar to autophagic cell death. However, some common features of PCD are highly conserved in both plants and metazoa.
The concept of “programmed cell-death” was used by Lockshin & Williams[4] in 1964 in relation to insect tissue development, around eight years before “apoptosis” was coined. Since then, PCD has become the more general of these two terms…
I’m a little disturbed at the bioenergetic researchers at Moscow State University because the concept of PCD applies to lower organisms, originally insects and lower, not humans.
I find their conjecture not naive, but uneasing considering that Russian black ops has the scalar weaponry for programming PCD into a living system where there was none before (see Death Transmission and Weapons that use Time Reversed EM Waves at the Bearden site – the concept of phase conjugates as the underlying foundation of reality perception is discussed at Nature’s Mind: The Quantum Hologram). I can only hope that the particular Russian researchers in this article are being naive.
As you probably already know, the human life span is unnaturally shortened through greedy industry practices in every sector of decadent society. Please have a close a look at the Poison Spectrum to understand the evil that is affecting everyone on this planet. Also read The Haig-Kissinger World Depopulation Policy.
